cell invasion

In vitro invasion assay was used to examine cell invasion capacity.

应用侵袭实验检测沉默Bmi-1基因表达后对A549细胞体外侵袭能力的影响。

Rac-1/ MMP-2 signaling pathway plays a positive role in tumor cell invasion and metastasis.

Rac1/MMP2通路在卵巢癌的侵袭转移中起着重要作用。

Role of tissue factor pathway inhibitor-2 in pancreatic cancer cell invasion

组织因子途径抑制物2对胰腺癌细胞浸润能力的影响

Integrin and MLCK are two key proteins involved in the process of tumor cell invasion and metastasis. The former regulates cell adhesion and the latter drives the cells to migrate.

整合素和MLCK是肿瘤细胞侵袭转移过程中调节细胞粘附稳定性和为细胞迁移提供驱动力的两个重要分子。

Objective: To investigate the expression of E-cadherin and Matrix metalloproteinase-2 ( MMP2) in human gastric cancer and their relationship with tumor cell invasion and metastasis.

目的探讨E-钙粘附素（E-cadherin）和基质金属蛋白酶-2（matrixmetalloporoteirnase-2，MMP-2）在胃癌组织中的表达与胃癌浸润转移的相关性。

Results: The in vitro cell invasion and adhesion potential of tumor cells from lymph node metastasis was significantly higher than those from primary tumor by 2.5 fold and 1.5 fold, respectively.

结果：淋巴结转移灶分离的肿瘤细胞的体外侵袭及粘附能力显著高于原发灶内分离的肿瘤细胞，大约分别为后者的2.5倍和1.5倍；

The process of implantation was similar, in some sense, to the process of tumor cell invasion and migration.

胚胎着床过程与肿瘤细胞的侵袭过程有一定的相似之处。

Although CD133+ tumor cells displayed an increased proliferation rate, they were not advantageous in cell invasion in vitro.

在体外，CD133+肝癌细胞具有较强的增殖能力，但并未表现出明显的侵袭方面的优势。

Background: Invasion and metastasis are major reasons for pancreatic cancer death. It will be of great significance to identify the molecules that specifically used by cancer cell invasion and its underlying molecular mechanism.

目的：浸润和转移是胰腺癌患者死亡的主要原因，探寻肿瘤细胞浸润转移的相关因子并阐明其分子机制对于寻找特异性的分子靶点以及胰腺癌的治疗具有重要意义。

Results and conclusion: Heparanase is the key enzyme to degrade extracellular matrix and closely related to tumor cell invasion and metastasis, become a novel cancer therapy target in gastrointestinal tumor.

结果与结论：乙酰肝素酶是降解细胞外基质的关键酶，与肿瘤细胞浸润转移密切相关，已成为消化道肿瘤治疗的新靶点。

Methods All19 patients suffering hepatic rupture were treated by arterial embolism, 8 cases by trauma and11 cases by liver cancer cell invasion.

方法经动脉栓塞治疗肝破裂出血19例，其中外伤性肝破裂8例，肝癌肝破裂11例。

There are no research between baicalein and endometrial cell invasion currently.

目前尚未有关于黄芩素和内异症在位内膜细胞侵袭性之间的研究。

The study of mitogen activated protein ( MAP) kinases signal pathway in host cell invasion by Toxoplasma gondii tachyzoites

刚地弓形虫速殖子侵入宿主细胞涉及的丝裂原蛋白激酶信号途径的研究

Objective To explore the role of extracellular matrix proteins in tumor cell invasion and metastasis.

目的探讨细胞外基质在肿瘤侵袭转移中的作用。

Studies on hepatic carcinoma cell invasion in vitro through Rho/ Rho kinase pathway

Rho/Rho激酶对肝癌细胞体外侵袭转移作用的研究

P53 expression and cancer cell invasion in the distal bowel wall of patients with rectal carcinoma

直肠癌远侧肠壁癌细胞的浸润及p53的表达

Cell invasion test was applied to observe the effect of lanthanum chloride on invasion.

细胞侵袭实验观察氯化镧对宫颈癌细胞侵袭能力的影响。

Bjective: To observe the angiogenesis of buccal carcinoma in early stage and the ultrastructural characters of new vascular and its relationship with carcinoma cell invasion.

目的：观察早期颊癌组织内血管生成现象，研究新生血管超微结构特征及其与癌细胞浸润生长的相互关系，探讨肿瘤内血管异常对颊癌局部生物学行为的影响。

Tumor cell invasion to the dura mater corresponding to dural tail sign occurs mainly in the 0.1-0.5 cm areas, partially in the regions of 0.6-1.0 cm, could not in the place beyond 1.0 cm.

肿瘤细胞侵袭脑膜尾征对应硬脑膜主要集中于0～0.5cm范围，少部分集中在0.6～1cm范围，无＞1.0cm者。

Over-expression of NF-κ B/ p65 was related to lymph node metastasis and depth of invasion, it may be related to cancer cell invasion and metastasis.

NF-κB/p65在有淋巴结转移和浸润至深肌层的宫颈癌组织中高表达，可能与肿瘤细胞侵袭转移有关。

But the specific mechanism of MIF promote tumor cell invasion and metastasis and its impact on biological behavior of cervical cancer cells by what pathways are not fully understood.

但MIF促进肿瘤细胞浸润转移的具体作用机制及其通过何种途径影响宫颈癌细胞的生物学行为尚不完全清楚。

No effects on cell invasion, mobility and adhesion were found in S2/ TXT cells treated with taxotere.

与SUIT-2细胞相比，TXT对S2/TXT细胞的侵袭、运动及黏附能力均无明显作用。

TIMP-1 Secreted by Fibroblasts Inhibits Tumor Cell Invasion and Metastasis in Mouse Melanoma

成纤维细胞分泌TIMP-1抑制小鼠黑色素瘤细胞浸润和转移

However, we found that miR-101-3p did not inhibit cancer cell invasion and migration as some literatures reported.

但是，发现miR-101-3p无抑制癌细胞侵袭和迁移的能力。

Gene expression of endoplasmic reticulum-retained anti-gelatinase intrabody and its inhibition of cancer cell invasion and proliferation

内质网滞留型抗明胶酶胞内抗体的基因表达及对肿瘤细胞侵袭和增殖的抑制作用

E-cadherin can suppress tumor cell invasion and metastasis, and is considered as an invasion/ metastasis suppressor gene.

E-cadherin能抑制肿瘤细胞的浸润和转移，被公认为是浸润、转移抑制基因。

Cepharanthine could inhibit tumor cell invasion and migration.

千金藤素能够抑制肿瘤细胞浸润和转移。

Conclusion Tumor cell invasion, vascular dilatation or proliferation, and connective tissue proliferation can lead to dural tail sign.

结论脑膜尾征对应硬脑膜的组织构成有三种：肿瘤细胞浸润，纤维结缔组织增生，及丰富的血管和血管扩张。

At the beginning, destruction of structural protein is regarded as the decisive step for cell invasion and migration.

起初认为结构蛋白的破坏是细胞侵袭、转移的决定性步骤。

Tumor cell invasion and metastasis is the main reason for the high cancer mortality rate, and cell invasion and metastasis is based on cell migration.

肿瘤细胞的浸润和转移是癌症高致死率的主要原因，而细胞浸润和转移的基础是细胞迁移。